中华医学会糖尿病分会全国委员(Committee Member of Chinese Diabetes Society, CMA)；
中华医学会骨质疏松与骨矿盐疾病分会常务委员（Standing Committee Member of Chinese Society of Osteoporosis and Bone Mineral Research, CMA）
揭示了PTH治疗骨质疏松的作用机制；发现骨细胞特异性膜蛋白E11能感受机械应力；优化了钙负荷PTH抑制试验；报告中国FOP的基因异常和临床表现；制备了ACVR1-Fc融合蛋白，证明了该融合蛋白可竞争性抑制FOP模型细胞的功能。
We revealed the mechanism of PTH effect on osteoporosis, found that E11 is an osteocyte-specific membrane protein which can sense mechanical stress on bone, and optimize the PTH suppression test by calcium load. We reported the genotype and phenotype of FOP patients in China, prepared the ACVR1-Fc fusion protein, and proved that this fusion protein could competitively suppress the function of FOP model cells.
